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Although the exact mechanism of cyclosporine nephrotoxicity remains unknown, its administration has been associated with in vivo reduction of GSH concentrations in the livers and kidneys of rats, which may be related to adverse effects of this immunosuppressive agent

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Similarly, OsWRKY45 expression was upregulated by 50%, 75%, 100%, and 150% in the respective treatments (Fig

Reversible glutathionylation of Complex I in response to oxidation of mitochondrial GSH pool has been shown to increase ROS formation [49]

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